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Airborne biogenic particles in the snow of the cities of the Russian Far East as potential allergic compounds.

https://arctichealth.org/en/permalink/ahliterature262711
Source
J Immunol Res. 2014;2014:141378
Publication Type
Article
Date
2014
Author
Kirill S Golokhvast
Source
J Immunol Res. 2014;2014:141378
Date
2014
Language
English
Publication Type
Article
Keywords
Air Pollutants - adverse effects - analysis
Allergens - adverse effects - analysis
Animals
Cities
Environmental monitoring
Far East
Humans
Particulate Matter - adverse effects - analysis
Risk factors
Russia
Snow
Abstract
This paper presents an analysis of airborne biogenic particles (1 mkm-1 mm) found in the snow in several cities of the Russian Far East during 2010-2013. The most common was vegetational terraneous detritus (fragments of tree and grass leaves) followed by animal hair, small insects and their fragments, microorganisms of aeroplankton, and equivocal biological garbage. Specific components were found in samples from locations close to bodies of water such as fragments of algae and mollusc shells and, marine invertebrates (needles of sea urchins and shell debris of arthropods). In most locations across the Far East (Vladivostok, Khabarovsk, Blagoveshchensk, and Ussuriysk), the content of biogenic particles collected in the winter did not exceed 10% of the total particulate matter, with the exception of Birobidzhan and the nature reserve Bastak, where it made up to 20%. Most of all biogenic compounds should be allergic: hair, fragments of tree and grass leaves, insects, and microorganisms.
Notes
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PubMed ID
25140327 View in PubMed
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Airborne particulate matter from primarily geologic, non-industrial sources at levels below National Ambient Air Quality Standards is associated with outpatient visits for asthma and quick-relief medication prescriptions among children less than 20 years old enrolled in Medicaid in Anchorage, Alaska.

https://arctichealth.org/en/permalink/ahliterature80178
Source
Environ Res. 2007 Mar;103(3):397-404
Publication Type
Article
Date
Mar-2007
Author
Chimonas Marc-Andre R
Gessner Bradford D
Author Affiliation
Division of Occupational and Environmental Medicine, Duke University Medical Center, Durham, NC 27710, USA. mchimonas@msn.com
Source
Environ Res. 2007 Mar;103(3):397-404
Date
Mar-2007
Language
English
Publication Type
Article
Keywords
Adolescent
Alaska - epidemiology
Ambulatory Care - statistics & numerical data
Anti-Asthmatic Agents - therapeutic use
Asthma - drug therapy - epidemiology - etiology
Child
Child, Preschool
Cohort Studies
Female
Humans
Infant
Infant, Newborn
Male
Medicaid
Models, Statistical
Particle Size
Particulate Matter - adverse effects - analysis - standards
Abstract
In Anchorage, Alaska, particulates with aerodynamic diameter or = 34 micro g/m(3). A significant 18.1% increase (RR: 1.181, 95% CI: 1.010-1.381) in the rate of quick-relief medication prescriptions occurred during days with PM(10) of 34-60 micro g/m(3), and a 28.8% increase (RR: 1.288, 95% CI: 1.026-1.619) occurred during days with PM(10) > or = 61 micro g/m(3). Similar results for outpatient asthma visits and quick-relief medication occurred in weekly models. There were no significant associations with PM(2.5) in either daily or weekly models. These subtle but statistically significant associations suggest that non-industrial, geologic sources of PM(10) may have measurable health effects at levels below current national standards.
PubMed ID
17049511 View in PubMed
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Ambient air pollution and primary liver cancer incidence in four European cohorts within the ESCAPE project.

https://arctichealth.org/en/permalink/ahliterature282438
Source
Environ Res. 2017 Apr;154:226-233
Publication Type
Article
Date
Apr-2017
Author
Marie Pedersen
Zorana J Andersen
Massimo Stafoggia
Gudrun Weinmayr
Claudia Galassi
Mette Sørensen
Kirsten T Eriksen
Anne Tjønneland
Steffen Loft
Andrea Jaensch
Gabriele Nagel
Hans Concin
Ming-Yi Tsai
Sara Grioni
Alessandro Marcon
Vittorio Krogh
Fulvio Ricceri
Carlotta Sacerdote
Andrea Ranzi
Ranjeet Sokhi
Roel Vermeulen
Kees de Hoogh
Meng Wang
Rob Beelen
Paolo Vineis
Bert Brunekreef
Gerard Hoek
Ole Raaschou-Nielsen
Source
Environ Res. 2017 Apr;154:226-233
Date
Apr-2017
Language
English
Publication Type
Article
Keywords
Air Pollutants - adverse effects - analysis
Air Pollution - adverse effects - analysis
Austria - epidemiology
Cohort Studies
Denmark - epidemiology
Environmental Exposure - adverse effects
Female
Humans
Incidence
Italy - epidemiology
Liver Neoplasms - epidemiology - etiology
Male
Nitrogen Oxides - adverse effects - analysis
Particulate Matter - adverse effects - analysis
Vehicle Emissions - analysis - toxicity
Abstract
Tobacco smoke exposure increases the risk of cancer in the liver, but little is known about the possible risk associated with exposure to ambient air pollution.
We evaluated the association between residential exposure to air pollution and primary liver cancer incidence.
We obtained data from four cohorts with enrolment during 1985-2005 in Denmark, Austria and Italy. Exposure to nitrogen oxides (NO2 and NOX), particulate matter (PM) with diameter of less than 10µm (PM10), less than 2.5µm (PM2.5), between 2.5 and 10µm (PM2.5-10) and PM2.5 absorbance (soot) at baseline home addresses were estimated using land-use regression models from the ESCAPE project. We also investigated traffic density on the nearest road. We used Cox proportional-hazards models with adjustment for potential confounders for cohort-specific analyses and random-effects meta-analyses to estimate summary hazard ratios (HRs) and 95% confidence intervals (CIs).
Out of 174,770 included participants, 279 liver cancer cases were diagnosed during a mean follow-up of 17 years. In each cohort, HRs above one were observed for all exposures with exception of PM2.5 absorbance and traffic density. In the meta-analysis, all exposures were associated with elevated HRs, but none of the associations reached statistical significance. The summary HR associated with a 10-µg/m(3) increase in NO2 was 1.10 (95% confidence interval (CI): 0.93, 1.30) and 1.34 (95% CI: 0.76, 2.35) for a 5-µg/m(3) increase in PM2.5.
The results provide suggestive evidence that ambient air pollution may increase the risk of liver cancer. Confidence intervals for associations with NO2 and NOX were narrower than for the other exposures.
PubMed ID
28107740 View in PubMed
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Assessment of the potential respiratory hazard of volcanic ash from future Icelandic eruptions: a study of archived basaltic to rhyolitic ash samples.

https://arctichealth.org/en/permalink/ahliterature287934
Source
Environ Health. 2017 Sep 11;16(1):98
Publication Type
Article
Date
Sep-11-2017
Author
David E Damby
Claire J Horwell
Gudrun Larsen
Thorvaldur Thordarson
Maura Tomatis
Bice Fubini
Ken Donaldson
Source
Environ Health. 2017 Sep 11;16(1):98
Date
Sep-11-2017
Language
English
Publication Type
Article
Keywords
Air Pollutants - adverse effects - analysis
Humans
Iceland
Particle Size
Particulate Matter - adverse effects - analysis
Silicates - adverse effects - analysis
Volcanic Eruptions - adverse effects - analysis
Abstract
The eruptions of Eyjafjallajökull (2010) and Grímsvötn (2011), Iceland, triggered immediate, international consideration of the respiratory health hazard of inhaling volcanic ash, and prompted the need to estimate the potential hazard posed by future eruptions of Iceland's volcanoes to Icelandic and Northern European populations.
A physicochemical characterization and toxicological assessment was conducted on a suite of archived ash samples spanning the spectrum of past eruptions (basaltic to rhyolitic magmatic composition) of Icelandic volcanoes following a protocol specifically designed by the International Volcanic Health Hazard Network.
Icelandic ash can be of a respirable size (up to 11.3 vol.% 
Notes
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PubMed ID
28893249 View in PubMed
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Association between air pollution from residential wood burning and dementia incidence in a longitudinal study in Northern Sweden.

https://arctichealth.org/en/permalink/ahliterature296869
Source
PLoS One. 2018; 13(6):e0198283
Publication Type
Journal Article
Research Support, Non-U.S. Gov't
Date
2018
Author
Anna Oudin
David Segersson
Rolf Adolfsson
Bertil Forsberg
Author Affiliation
Occupational and Environmental Medicine, Department of Public Health and Clinical Medicine, Umeå University, Umeå, Sweden.
Source
PLoS One. 2018; 13(6):e0198283
Date
2018
Language
English
Publication Type
Journal Article
Research Support, Non-U.S. Gov't
Keywords
Adult
Age Distribution
Aged
Aged, 80 and over
Air Pollution - adverse effects - analysis
Dementia - chemically induced - epidemiology
Female
Fires
Housing
Humans
Incidence
Longitudinal Studies
Male
Middle Aged
Particulate Matter - adverse effects - analysis
Risk factors
Sweden - epidemiology
Vehicle Emissions - analysis
Wood - adverse effects - chemistry
Abstract
There is highly suggestive evidence for an effect of air pollution exposure on dementia-related outcomes, but evidence is not yet present to clearly pinpoint which pollutants are the probable causal agents. The aims of this study was to assess the longitudinal association between exposures of fine ambient particulate matter (PM2.5) from residential wood burning, and vehicle exhaust, with dementia.
We used data from the Betula study, a longitudinal study of dementia in Umeå, Northern Sweden. The study size was 1 806 and the participants were followed from study entry (1993-1995) to 2010. Modelled levels of source-specific fine particulate matter at the residential address were combined with information on wood stoves or wood boilers, and with validated data on dementia diagnosis and individual-level characteristics from the Betula study. Cox proportional hazards models were used to estimate Hazard Ratios (HRs) and their 95% CIs for dementia incidence (vascular dementia and Alzheimer's disease), adjusted for individual-level characteristics.
The emission of PM2.5 from local residential wood burning was associated with dementia incidence with a hazard ratio of 1.55 for a 1 µg/m3 increase in PM2.5 (95% Confidence Interval (CI): 1.00-2.41, p-value 0.05). Study participants with an address in an area with the highest quartile of PM2.5 from residential wood burning and who also had a wood-burning stove were more likely to develop dementia than those in the lower three quartiles without a wood-burning stove with hazard ratios of 1.74 (CI: 1.10-2.75, p-value 0.018). Particulate matter from traffic exhaust seemed to be associated with dementia incidence with hazard ratios of 1.66, (CI: 1.16-2.39), p-value 0.006, and 1.41 (CI: 0.97-2.23), p-value 0.07, in the third and fourth quartiles, respectively.
If the associations we observed are causal, then air pollution from residential wood burning, and air pollution from traffic, might be independent important risk factors for dementia.
PubMed ID
29897947 View in PubMed
Less detail

Coarse and fine particles but not ultrafine particles in urban air trigger hospital admission for asthma in children.

https://arctichealth.org/en/permalink/ahliterature128939
Source
Thorax. 2012 Mar;67(3):252-7
Publication Type
Article
Date
Mar-2012
Author
Amne Iskandar
Zorana Jovanovic Andersen
Klaus Bønnelykke
Thomas Ellermann
Klaus Kaae Andersen
Hans Bisgaard
Author Affiliation
Copenhagen Prospective Studies on Asthma in Childhood, Health Sciences, University of Copenhagen, Copenhagen University Hospital, Ledreborg Allé 34, 2820 Gentofte, Denmark.
Source
Thorax. 2012 Mar;67(3):252-7
Date
Mar-2012
Language
English
Publication Type
Article
Keywords
Adolescent
Age Factors
Asthma - epidemiology - etiology
Child
Child, Preschool
Cross-Over Studies
Denmark - epidemiology
Environmental Monitoring - methods
Epidemiological Monitoring
Hospitalization - statistics & numerical data
Humans
Infant
Infant, Newborn
Nitrogen Dioxide - adverse effects - analysis
Nitrogen Oxides - adverse effects - analysis
Particle Size
Particulate Matter - adverse effects - analysis
Sex Factors
Urban Health - statistics & numerical data
Abstract
Short-term exposure to air pollution can trigger hospital admissions for asthma in children, but it is not known which components of air pollution are most important. There are no available studies on the particular effect of ultrafine particles (UFPs) on paediatric admissions for asthma.
To study whether short-term exposure to air pollution is associated with hospital admissions for asthma in children. It is hypothesised that (1) the association between asthma admissions and air pollution is stronger with UFPs than with coarse (PM10) and fine (PM2.5) particles, nitrogen oxides (NOx) or nitrogen dioxide (NO2); and (2) infants are more susceptible to the effects of exposure to air pollution than older children.
Daily counts of admissions for asthma in children aged 0-18 years to hospitals located within a 15 km radius of the central fixed background urban air pollution measurement station in Copenhagen between 2001 and 2008 were extracted from the Danish National Patient Registry. A time-stratified case crossover design was applied and data were analysed using conditional logistic regression to estimate the effect of air pollution on asthma admissions.
A significant association was found between hospital admissions for asthma in children aged 0-18 years and NOx (OR 1.11; 95% CI 1.05 to 1.17), NO2 (1.10; 95% CI 1.04 to 1.16), PM10 (1.07; 95% CI 1.03 to 1.12) and PM2.5 (1.09; 95% CI 1.04 to 1.13); there was no association with UFPs. The association was stronger in infants than in older children for all pollutants, but no statistically significant interaction was detected.
Short-term exposure to air pollution can trigger hospital admission for asthma in children, with infants possibly being most susceptible. These effects seemed to be mediated by larger particles and traffic-related gases, whereas UFPs showed no effect.
PubMed ID
22156960 View in PubMed
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Coarse Fraction Particle Matter and Exhaled Nitric Oxide in Non-Asthmatic Children.

https://arctichealth.org/en/permalink/ahliterature283704
Source
Int J Environ Res Public Health. 2016 Jun 22;13(6)
Publication Type
Article
Date
Jun-22-2016
Author
Hanne Krage Carlsen
Peter Boman
Bodil Björ
Anna-Carin Olin
Bertil Forsberg
Source
Int J Environ Res Public Health. 2016 Jun 22;13(6)
Date
Jun-22-2016
Language
English
Publication Type
Article
Keywords
Air Pollutants - analysis
Child
Exhalation - physiology
Female
Humans
Inflammation - etiology - physiopathology
Male
Models, Theoretical
Nitric Oxide - analysis
Particulate Matter - adverse effects - analysis
Sweden
Abstract
Coarse particle matter, PMcoarse, is associated with increased respiratory morbidity and mortality. The aim of this study was to investigate the association between short-term changes in PMcoarse and sub-clininal airway inflammation in children. Healthy children aged 11 years from two northern Swedish elementary schools underwent fraction of exhaled nitrogen oxide (FENO) measurements to determine levels of airway inflammation twice weekly during the study period from 11 April-6 June 2011. Daily exposure to PMcoarse, PM2.5, NO2, NOx, NO and O3 and birch pollen was estimated. Multiple linear regression was used. Personal covariates were included as fixed effects and subjects were included as a random effect. In total, 95 children participated in the study, and in all 493 FENO measurements were made. The mean level of PMcoarse was 16.1 µg/m³ (range 4.1-42.3), and that of O3 was 75.0 µg/m³ (range: 51.3-106.3). That of NO2 was 17.0 µg/m³ (range: 4.7-31.3), NOx was 82.1 µg/m³ (range: 13.3-165.3), and NO was 65 µg/m³ (range: 8.7-138.4) during the study period. In multi-pollutant models an interquartile range increase in 24 h PMcoarse was associated with increases in FENO by between 6.9 ppb (95% confidence interval 0.0-14) and 7.3 ppb (95% confidence interval 0.4-14.9). PMcoarse was associated with an increase in FENO, indicating sub-clinical airway inflammation in healthy children.
Notes
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PubMed ID
27338437 View in PubMed
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Controlling occupational allergies in the workplace.

https://arctichealth.org/en/permalink/ahliterature162395
Source
Int J Occup Med Environ Health. 2007;20(2):107-15
Publication Type
Article
Date
2007
Author
Anne Korpi
Sanna Lappalainen
Eila Kaliste
Pentti Kalliokoski
Kari Reijula
Anna-Liisa Pasanen
Author Affiliation
Department of Environmental Science, University of Kuopio, Kuopio, Finland.
Source
Int J Occup Med Environ Health. 2007;20(2):107-15
Date
2007
Language
English
Publication Type
Article
Keywords
Allergens - adverse effects - analysis
Asthma - chemically induced - epidemiology - prevention & control
Environmental Monitoring - methods
Epidemiological Monitoring
Female
Finland - epidemiology
Humans
Hypersensitivity - epidemiology - prevention & control
Inhalation Exposure - adverse effects - prevention & control
Interviews as Topic
Male
Observation
Occupational Exposure - adverse effects - prevention & control
Particulate Matter - adverse effects - analysis
Prevalence
Questionnaires
Risk Assessment - methods
Safety
Safety Management - methods
Abstract
In recent years, the prevalence of work-related asthma has increased. Therefore, more attention needs to be paid to occupational allergens and their avoidance and control in workplaces. However, risk assessment of occupational allergen exposure is difficult because the relationship between exposure concentration, sensitization, and symptoms has not been fully established. This paper introduces a systematic and comprehensive approach to assessing and managing allergen risks at workplaces.
This approach relies on the cooperation and active communication during the whole process between management, employees, and health care personnel, with the assistance of experts when needed. In addition to gathering background information, including allergic symptoms, through questionnaires addressed to the management and employees, hazard identification is also processed in the workplace through observations and measurements. The methods generally recommended to reduce allergen exposure are compared with those used in the workplace. The process is to be carefully planned and documented to allow later follow-up and re-evaluation.
The multi-faceted approach encompasses several risk assessment techniques, and reveals the prevalence of work-related allergic symptoms. The process effectively focuses on the potential means for controlling allergen exposure.
Based on this approach, the synopsis on the critical points that require implementation of effective control measures can be presented.
PubMed ID
17638677 View in PubMed
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Ecological analysis of long-term exposure to ambient air pollution and the incidence of stroke in Edmonton, Alberta, Canada.

https://arctichealth.org/en/permalink/ahliterature142950
Source
Stroke. 2010 Jul;41(7):1319-25
Publication Type
Article
Date
Jul-2010
Author
Julie Y M Johnson
Brian H Rowe
Paul J Villeneuve
Author Affiliation
Air Health Effects Division, Health Canada, University of Alberta, Edmonton, Canada.
Source
Stroke. 2010 Jul;41(7):1319-25
Date
Jul-2010
Language
English
Publication Type
Article
Keywords
Air Pollution - adverse effects - analysis
Alberta - epidemiology
Ecology - methods
Female
Humans
Incidence
Inhalation Exposure - adverse effects - analysis
Male
Particulate Matter - adverse effects - analysis
Socioeconomic Factors
Stroke - epidemiology - etiology
Time Factors
Abstract
Long-term air pollution effects on stroke incidence have not been examined extensively. We investigated the associations between ambient pollution and the incidence of stroke, as well as stroke subtypes, in a northern Canadian city surrounded by energy-sector pollution sources.
Stroke data from an administrative database from 2003 through 2007 were used to estimate annual incidence rates within small geographic regions within Edmonton, Canada. Air pollution levels for each region were estimated from continuous fixed-site monitoring stations in and around Edmonton. We fit models estimating stroke risk in relation to pollution levels; risks were adjusted for age, sex, income, social deprivation, and other factors.
Between 2003 and 2007, the average 5-year concentration of NO(2) and CO was positively associated with the incidence of stroke, particularly for hemorrhagic and nonhemorrhagic stroke subtypes (NO(2): hemorrhagic stroke relative risk=1.46; 95% CI, 1.19-1.80; nonhemorrhagic stroke relative risk=1.36; 95% CI, 1.19-1.56). However, these estimates of risk diminished after controlling for the ecological measures of income and deprivation. Adjustment for ecologically derived indices of smoking, hypertension, and body mass index did not alter the estimates of risk in any meaningful way.
Although long-term NO(2) and CO levels were positively associated with a higher incidence of stroke in the entire study area, the risk estimates were strongly attenuated by household income levels. Further research that incorporates individual-level risk factor data would improve our understanding of the relation of longer-term exposures to ambient air pollution and stroke outcomes.
PubMed ID
20538697 View in PubMed
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Effects of concentrated ambient particles and diesel engine exhaust on allergic airway disease in Brown Norway rats.

https://arctichealth.org/en/permalink/ahliterature97836
Source
Res Rep Health Eff Inst. 2009 Nov;(145):5-55
Publication Type
Article
Date
Nov-2009
Author
Jack R Harkema
James G Wagner
Norbert E Kaminski
Masako Morishita
Gerald J Keeler
Jacob D McDonald
Edward G Barrett
Author Affiliation
Department of Pathobiology and Diagnostic Investigation, Michigan State University, East Lansing, Michigan 48864, USA. harkemaj@msu.edu
Source
Res Rep Health Eff Inst. 2009 Nov;(145):5-55
Date
Nov-2009
Language
English
Publication Type
Article
Keywords
Air Pollutants - adverse effects - analysis
Allergens
Animals
Asthma
Bronchial Hyperreactivity - diagnosis - etiology - pathology
Male
Particulate Matter - adverse effects - analysis
Rats
Vehicle Emissions - toxicity
Abstract
Increased concentrations of airborne fine particulate matter (PM2.5; particulate matter with an aerodynamic diameter
PubMed ID
20198910 View in PubMed
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25 records – page 1 of 3.