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173 records – page 1 of 18.

[A correlational analysis of the morphometric cardiac indices in virtually healthy persons and in patients with ischemic heart disease and arterial hypertension in different age groups]

https://arctichealth.org/en/permalink/ahliterature54899
Source
Lik Sprava. 1994 May-Jun;(5-6):111-4
Publication Type
Article

Acute necrotizing encephalopathy in caucasian children: two cases and review of the literature.

https://arctichealth.org/en/permalink/ahliterature174029
Source
J Child Neurol. 2005 Jun;20(6):527-32
Publication Type
Article
Date
Jun-2005
Author
Adam Kirton
Kevin Busche
Catherine Ross
Elaine Wirrell
Author Affiliation
Division of Pediatric Neurology, Department of Pediatrics, University of Calgary, Calgary, Alberta, Canada.
Source
J Child Neurol. 2005 Jun;20(6):527-32
Date
Jun-2005
Language
English
Publication Type
Article
Keywords
Brain - pathology
Canada
Chickenpox
European Continental Ancestry Group
Fatal Outcome
Female
Humans
Infant
Influenza A virus
Influenza, Human
Leukoencephalitis, Acute Hemorrhagic - etiology - pathology
Liver Diseases - etiology
Myocardium - pathology
Necrosis
Rotavirus Infections
Seizures - etiology
Tomography, X-Ray Computed
Abstract
Acute necrotizing encephalopathy is a fulminant neurologic disease seen predominantly in Japan and Taiwan. We present two cases diagnosed at a Canadian center within the same year in Caucasian children. Both were previously well, developed an acute viral illness with fever and vomiting, and progressed to brain death within 2 to 4 days. Neuroimaging and postmortem examination demonstrated the unique features of bilateral and severe necrosis of deep gray- and subcortical white-matter structures. The first case was associated with extensive, but transient, hepatic involvement, recent varicella and rotavirus infections, and detailed metabolic studies, including mitochondrial functional analysis, were normal. The second case tested positive for influenza A infection, whereas evidence of liver damage was lacking. Both children demonstrated early lymphopenia and myocardial necrosis, two features not previously associated with acute necrotizing encephalopathy. These cases are unique in their occurrence in non-Japanese children and are among the first published reports in Canada.
PubMed ID
15996405 View in PubMed
Less detail

Adaptive registration of varying contrast-weighted images for improved tissue characterization (ARCTIC): application to T1 mapping.

https://arctichealth.org/en/permalink/ahliterature268568
Source
Magn Reson Med. 2015 Apr;73(4):1469-82
Publication Type
Article
Date
Apr-2015
Author
Sébastien Roujol
Murilo Foppa
Sebastian Weingärtner
Warren J Manning
Reza Nezafat
Source
Magn Reson Med. 2015 Apr;73(4):1469-82
Date
Apr-2015
Language
English
Publication Type
Article
Keywords
Algorithms
Artifacts
Female
Humans
Image Enhancement - methods
Image Interpretation, Computer-Assisted - methods
Magnetic Resonance Imaging, Cine - methods
Male
Middle Aged
Motion
Myocardium - pathology
Pattern Recognition, Automated - methods
Reproducibility of Results
Sensitivity and specificity
Subtraction Technique
Abstract
To propose and evaluate a novel nonrigid image registration approach for improved myocardial T1 mapping.
Myocardial motion is estimated as global affine motion refined by a novel local nonrigid motion estimation algorithm. A variational framework is proposed, which simultaneously estimates motion field and intensity variations, and uses an additional regularization term to constrain the deformation field using automatic feature tracking. The method was evaluated in 29 patients by measuring the DICE similarity coefficient and the myocardial boundary error in short axis and four chamber data. Each image series was visually assessed as "no motion" or "with motion." Overall T1 map quality and motion artifacts were assessed in the 85 T1 maps acquired in short axis view using a 4-point scale (1-nondiagnostic/severe motion artifact, 4-excellent/no motion artifact).
Increased DICE similarity coefficient (0.78 ± 0.14 to 0.87 ± 0.03, P
Notes
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Cites: Magn Reson Med. 2014 Feb;71(2):823-923440632
Cites: Herz. 2000 Jun;25(4):384-9110948774
PubMed ID
24798588 View in PubMed
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[A fatal case of chronic Q fever with cardiovascular involvement]

https://arctichealth.org/en/permalink/ahliterature54740
Source
Lik Sprava. 1995 Sep-Dec;(9-12):170-3
Publication Type
Article
Author
Z H Kushnir
Iu A Ivaniv
B I Ribun
O A Khoma
Source
Lik Sprava. 1995 Sep-Dec;(9-12):170-3
Language
Ukrainian
Publication Type
Article
Keywords
Adult
Chronic Disease
Endocarditis, Bacterial - etiology - pathology
English Abstract
Fatal Outcome
Heart Failure, Congestive - etiology - pathology
Humans
Male
Myocardium - pathology
Q Fever - complications - pathology
Rheumatic Heart Disease - complications - pathology
Abstract
A lethal case is reported of chronic Q-fever in a patient aged 34 who regarded himself as completely healthy six months before his death. The diagnosis was made on the basis of vital investigation of blood sera in a complement-fixation test and indirect fluorescent-antibody test (antibody titers 1 : 1280 against phase 1 and 1 : 320 against phase 2 Coxiella burnetii), differentiation of antibodies as distinct classes of immunoglobulins, results of pathoanatomical and microbiological investigations. Pathoanatomical features of the organs are fully detailed, especially those of the heart. Death occurred in the presence of cardiac failure growing progressively worse, involving many organs. Patients with cardiovascular pathology have to be examined for Q-fever in order that we should be able to early detect and apply specific therapy to treat its chronic form.
PubMed ID
8983769 View in PubMed
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Age-associated changes in hearts of male Fischer 344/Brown Norway F1 rats.

https://arctichealth.org/en/permalink/ahliterature79665
Source
Ann Clin Lab Sci. 2006;36(4):427-38
Publication Type
Article
Date
2006
Author
Walker Ernest M
Nillas Michael S
Mangiarua Elsa I
Cansino Sylvestre
Morrison Ryan G
Perdue Romaine R
Triest William E
Wright Gary L
Studeny Mark
Wehner Paulette
Rice Kevin M
Blough Eric R
Author Affiliation
Department of Pathology, Joan C Edwards School of Medicine, Marshall University, Huntington, WV 25704, USA. walkere@marshall.edu
Source
Ann Clin Lab Sci. 2006;36(4):427-38
Date
2006
Language
English
Publication Type
Article
Keywords
Aging - physiology
Animals
Arrhythmia - pathology - physiopathology
Disease Models, Animal
Echocardiography - methods
Heart - physiopathology
Heart Diseases - pathology - physiopathology
Heart Ventricles - pathology - physiopathology
Hypertrophy, Left Ventricular - pathology - physiopathology
Male
Myocardium - pathology
Organ Size
Rats
Rats, Inbred BN
Rats, Inbred F344
Ventricular Dysfunction, Left - pathology - physiopathology
Abstract
Aging is associated with left ventricular hypertrophy, dilatation, and fibrosis of the heart. The Fischer 344/Brown Norway F1 (F344/BNF1) rat is recommended for age-related studies by the National Institutes on Aging because this hybrid rat lives longer and has a lower rate of pathological conditions than inbred rats. However, little is known about age-associated changes in cardiac and aortic function and structure in this model. This study evaluated age-related cardiac changes in male F344/BNF1 rats using ECHO, gross, and microscopic examinations. Rats aged 6-, 30-, and 36-mo were anesthetized and two-dimensional ECHO measurements, two-dimensional guided M-mode, Doppler M-mode, and other recordings from parasternal long- and short-axis views were obtained using a Phillips 5500 ECHO system with a 12 megahertz transducer. Hearts and aortas from sacrificed rats were evaluated grossly and microscopically. The ECHO studies revealed persistent cardiac arrhythmias (chiefly PVCs) in 72% (13/18) of 36-mo rats, 10% (1/10) of 30-mo rats, and none in 6-mo rats (0/16). Gross and microscopic studies showed left ventricular (LV) dilatation, borderline to mild hypertrophy, and areas of fibrosis that were common in 36-mo rats, less evident in 30-mo rats, and absent in 6-mo rats. Aging was associated with mild to moderate decreases of LV diastolic and systolic function. Thus, male F344/BN F1 rats demonstrated progressive age-related (a) decline in cardiac function (diastolic and systolic indices), (b) LV structural changes (chamber dimensions, volumes, and wall thicknesses), and (c) persistent arrhythmias. These changes are consistent with those in humans. The noninvasive ECHO technique offers a means to monitor serial age-related cardiac failure and therapeutic responses in the same rats over designated time intervals.
PubMed ID
17127729 View in PubMed
Less detail

Alterative and plastic insufficiency of cardiomyocytes: isoproterenol-induced damage to myocardium during anthracycline cardiomyopathy.

https://arctichealth.org/en/permalink/ahliterature10133
Source
Bull Exp Biol Med. 2001 Jun;131(6):589-94
Publication Type
Article
Date
Jun-2001
Author
E L Lushnikova
L M Nepomnyashchikh
D E Semenov
Author Affiliation
Department of General Pathology and Pathomorphology, Institute of Regional Pathology and Pathomorphology, Siberian Division of the Russian Academy of Medical Sciences, Novosibirsk. pathol@cyber.nsk.ma.ru
Source
Bull Exp Biol Med. 2001 Jun;131(6):589-94
Date
Jun-2001
Language
English
Publication Type
Article
Keywords
Animals
Antibiotics, Antineoplastic - toxicity
Cardiomyopathies - chemically induced - pathology - physiopathology
Cardiotonic Agents - pharmacology
Cell Death - drug effects
Daunorubicin - toxicity
Isoproterenol - pharmacology
Myocardial Contraction - drug effects
Myocardium - pathology
Rats
Rats, Wistar
Abstract
The development of regenerative and plastic myocardial insufficiency induced by anthracycline antibiotic rubomycin is accompanied by a decrease in cardiomyocyte sensitivity to damage produced by synthetic catecholamine isoproterenol. The incidence and the size of coagulation necrosis foci of cardiomyocytes developed 6 h after isoproterenol injection significantly decreased with increasing in the interval between rubomycin injection and subsequent administration of isoproterenol. In Wistar rats receiving rubomycin 3-5 days prior to isoproterenol and exhibiting signs of regenerative and plastic insufficiency, no cardiomyocyte contracture, intracellular myocytolysis, or lump degradation characteristic of cardiac insufficiency induced by endo- and exogenous catecholamines were found.
PubMed ID
11586415 View in PubMed
Less detail

[A matter of heart--two examinations of the same heart with a sixty year interval]

https://arctichealth.org/en/permalink/ahliterature53967
Source
Tidsskr Nor Laegeforen. 2000 Dec 10;120(30):3699-701
Publication Type
Article
Date
Dec-10-2000
Author
J. Grande
Author Affiliation
Institutt for tverrfaglige kulturstudier, Norges teknisk-naturvitenskapelige universitet, 7491 Trondheim. jan.grande@hf.ntnu.no
Source
Tidsskr Nor Laegeforen. 2000 Dec 10;120(30):3699-701
Date
Dec-10-2000
Language
Norwegian
Publication Type
Article
Keywords
Cardiology - history
Endocarditis, Bacterial - history - microbiology - pathology
English Abstract
History, 19th Century
History, 20th Century
Humans
Myocardium - pathology
Norway
Portraits
Abstract
The 20th of May 1869, Professor Emanuel Winge used a human heart as an exhibit at a meeting of The Medical Society in Christiania. This heart was later conserved and kept in the museum of the Institute of Pathology. Sixty years later, one of Winge's successors, Professor Francis Harbitz, used the very same heart, also at a meeting of The Medical Society. Harbitz had then confirmed Winge's hypothesis of 1869, that the endocarditis of this heart was due to a bacterial infection. This article contemplates the disparity between the "scientific gazes" of Winge and Harbitz, and why Harbitz was able to convert Winges hypothesis of 1869 into a scientific discovery in 1929.
PubMed ID
11215941 View in PubMed
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[Analysis of lethal outcomes of rheumatic diseases in Moscow].

https://arctichealth.org/en/permalink/ahliterature184598
Source
Ter Arkh. 2003;75(5):78-82
Publication Type
Article
Date
2003
Author
Sh Erdes
A B Demina
O M Folomeeva
S G Radenska-Lopovok
O V Zairat'iants
Source
Ter Arkh. 2003;75(5):78-82
Date
2003
Language
Russian
Publication Type
Article
Keywords
Autopsy
Bone Diseases - mortality
Cause of Death
Humans
Middle Aged
Moscow
Muscular Diseases - mortality
Myocardium - pathology
Rheumatic Diseases - mortality - pathology
Rheumatic Fever - mortality - pathology
Rheumatic Heart Disease - mortality - pathology
Russia
Abstract
To study the pattern of lethal outcomes due to rheumatic diseases (RD) in Moscow.
Annual reports of 38 pathological departments of Moscow have been analysed for 1999 and 2000.
RD accounted for 1.8% diagnosis at autopsies (n = 784). RD as the main diagnosis was in 668 cases (1.53%). Diseases of the bone-muscle system caused death 3.5 times less often than rheumatism. As concommitent diseases RD were encountered in 118 cases (0.27%), diseases of the bone-muscle system were registered 2 times less frequently than rheumatism. Chronic rheumatic diseases of the heart were diagnosed in 590 cases (98.5%), rheumatic fever was detected in 9 (1.5%) patients. The main diagnosis of RA, seronegative arthritides, systemic vasculitides, SLE, osteoarthrosis was made in 49, 10 9.3, 12.7, 1.3%, respectively. Such nosological entities as osteoporosis, gout and other microcrystalline arthritides were referred to the group "other rheumatic diseases" and made up 12.7%. As concomitant pathology RA, OA, seronegative spondyloarthritides, SLE, other RD occurred in 54, 8.1, 27, 2.7, 2.7%, respectively.
The share of RD in autopsy diagnosis accounts for 1.8% of the total number of necropsies. These figures seem to underestimate the real situation and may be explained by poor registration of RD at autopsy and a fall in the total number of autopsies for the last 10 years. For Moscow and Russia as a whole there is a prevalence of rheumatism mortality (76%), primarily deaths of chronic rheumatic cardiac diseases, over mortality due to diseases of the bone-muscular system (24%).
PubMed ID
12847905 View in PubMed
Less detail

Analysis of sudden unexpected death in southern Ontario, with emphasis on myocarditis.

https://arctichealth.org/en/permalink/ahliterature247382
Source
Can Med Assoc J. 1979 Mar 17;120(6):676-80, 706
Publication Type
Article
Date
Mar-17-1979
Author
P. Wentworth
L A Jentz
A E Croal
Source
Can Med Assoc J. 1979 Mar 17;120(6):676-80, 706
Date
Mar-17-1979
Language
English
Publication Type
Article
Keywords
Adolescent
Adult
Coxsackievirus Infections - complications - microbiology
Death, Sudden - etiology - pathology
Enterovirus - isolation & purification
Female
Humans
Male
Middle Aged
Myocarditis - complications - microbiology - pathology
Myocardium - pathology
Ontario
Retrospective Studies
Abstract
The records of all 2427 autopsies performed at the Brantford (Ont.) General and Paris (Ont.) Willett hospitals from Jan. 1, 1969 to Aug. 15, 1978 were reviewed. Of the 1299 cases of sudden unexpected death investigated by a coroner almost 28% were due to unnatural causes--violence or poisoning. The main cause of natural sudden death was coronary artery disease, which accounted for 43.3% of all the sudden unexpected deaths. In 20 cases the cause of death was thought to be viral myocarditis, and in 9 of the 20 there was serologic evidence of at least previous coxsackievirus disease. Two of the nine cases were of special interest because of the finding of giant-cell myocarditis in one and aortic valve disease in the other. Eleven of the 20 persons were aged 13 to 46 years. These findings support the view that the most serious manifestation of enterovirus infection today is cardiac damage by coxsackieviruses.
Notes
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PubMed ID
436050 View in PubMed
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Analysis of the results of the cooperative study between the Granada and Copenhagen University Institutes of Forensic Medicine.

https://arctichealth.org/en/permalink/ahliterature55461
Source
Acta Med Leg Soc (Liege). 1989;39(1):199-202
Publication Type
Article
Date
1989

173 records – page 1 of 18.