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30 records – page 1 of 3.

The assessment of exposure in terms of fibres.

https://arctichealth.org/en/permalink/ahliterature217636
Source
Ann Occup Hyg. 1994 Aug;38(4):477-87, 409-10
Publication Type
Article
Date
Aug-1994
Author
G W Gibbs
Source
Ann Occup Hyg. 1994 Aug;38(4):477-87, 409-10
Date
Aug-1994
Language
English
Publication Type
Article
Keywords
Asbestos, Serpentine - adverse effects
Asbestosis
Canada
Dust
Environmental monitoring
Great Britain
Humans
Lung Neoplasms - etiology
Mining
Occupational Exposure - analysis
Textile Industry
United States
Abstract
The membrane filter (MF) method for evaluating asbestos fibre concentrations was introduced in the 1960s. Before that time the midget impinger (MI) was used in North America, while the long running (LRTP) and regular thermal precipitator (TP) were used in the U.K. All studies from which estimates of long-term health risks can be derived (i.e. those with individual cumulative lifetime exposure estimates) were based on the now obsolete methods. The reliability of converting these indices of exposure to MF equivalent concentrations was reviewed. It was concluded that no overall single factor could be derived for the Quebec mining and milling industry. However, it has been possible to derive conversion factors at the individual mill and work area level. Applying these in one Quebec mortality study analysis based on all jobs held by persons in the cohort gave an overall MF/MI ratio of 3.6. An examination of the confidence intervals surrounding the Quebec data, ratios derived for other chrysotile mines by other investigators, and measurements of fibre concentrations in the 1970s suggest that this was probably not unreasonable. Side-by-side and other measurements were used to convert MI concentrations in the U.S. textile industry to MF fibre concentrations. While conversions involve considerable uncertainty, independent measurements of fibres in the lung tissues of workers from the U.S. textile plant and Quebec mills show that in lungs the ratios of the concentrations of chrysotile to those of tremolite are quite consistent with the ratio of assessed exposures to these fibres in the two industries. There is an apparently higher risk of mesothelioma in one Quebec mining area (Thetford Mines) than in another (Asbestos). A high concentration of fibrous tremolite has been found in the lungs of workers in Thetford. A method of evaluating the extent to which mesothelioma risk in the chrysotile mining industry might be explained by tremolite exposures was proposed. The slope of the lung cancer dose-response relationship for the textile industry is approximately 50 times that for the mining and milling industry. Available data on the length distributions of fibres from Quebec mines and mills (up to 5% > 5 microns) and the Charleston textile plant (up to 21% > 5 microns) and some marginal indication of longer fibres in tissues from Charleston workers suggest that further work specifically addressing differences in the size distributions of long fibres in these industries is needed.
PubMed ID
7978969 View in PubMed
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[Biologic markers for early diagnosis of effects caused by exposure to coal dust in miners].

https://arctichealth.org/en/permalink/ahliterature118928
Source
Med Tr Prom Ekol. 2012;(9):36-42
Publication Type
Article
Date
2012
Author
N A Pavlovskaia
O P Rushkevich
Source
Med Tr Prom Ekol. 2012;(9):36-42
Date
2012
Language
Russian
Publication Type
Article
Keywords
Biological Markers - analysis
Coal
Coal Mining
Dust
Early Diagnosis
Humans
Incidence
Male
Occupational Diseases - diagnosis - epidemiology - metabolism
Respiration Disorders - chemically induced - diagnosis - metabolism
Russia - epidemiology
Abstract
The authors studied changes in several laboratory values of coal miners in Russian Federation, defined information value of these changes and suggested complex of methods for early preclinical diagnosis of negative effects caused by coal dust in the miners. Dust-related respiratory diseases were proved to develop by stages on molecular level.
PubMed ID
23156063 View in PubMed
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[Changes in dust content of workplace air during ore extraction at the Bazhenovsk chrysotile asbestos field].

https://arctichealth.org/en/permalink/ahliterature181738
Source
Med Tr Prom Ekol. 2003;(12):39-42
Publication Type
Article
Date
2003

[Characteristics of the combined action of the beryllium-containing dust from complex metal ores in the Far North (clinical hygienic and experimental studies)].

https://arctichealth.org/en/permalink/ahliterature246864
Source
Gig Tr Prof Zabol. 1979 Sep;(9):15-9
Publication Type
Article
Date
Sep-1979

[Characteristics of the pathological degree of tooth wear in coal mine workers]

https://arctichealth.org/en/permalink/ahliterature62982
Source
Stomatologiia (Mosk). 1980 Mar-Apr;59(2):53-5
Publication Type
Article

[Chronic occupational bronchitis in workers of coal extracting enterprises in Kouzbass: role of endogenous factors].

https://arctichealth.org/en/permalink/ahliterature143489
Source
Med Tr Prom Ekol. 2010;(3):37-40
Publication Type
Article
Date
2010
Author
N I Gafarov
V V Zakharenkov
N I Panev
A V Burdein
V P Puzyrev
A A Rudko
Source
Med Tr Prom Ekol. 2010;(3):37-40
Date
2010
Language
Russian
Publication Type
Article
Keywords
Acid Phosphatase - blood
Adult
Biological Markers - blood
Bronchitis, Chronic - blood - epidemiology - genetics
Carboxylesterase - blood
Coal
Coal Mining
Dust
Genetic Predisposition to Disease
Genotype
Haptoglobins - metabolism
Humans
Middle Aged
Occupational Diseases - blood - epidemiology - genetics
Occupational Exposure - adverse effects
Polymorphism, Genetic
Prevalence
Siberia - epidemiology
Vitamin D-Binding Protein - blood
Abstract
The authors studied distribution of biochemical markers for HP, GC, EsD, AcP genes, polymorphism of GSTT1 (GST-theta 1), GSTM1 (GST-mu 1), locus WNTR of NOS3 gene (alleles A/B) in chronic dust bronchitis patients and in apparently healthy individuals. Genotypes EsD 1-2 and AcP bb individuals were proved to be most prone to the disease. Endogenous resistent factors for chronic dust bronchitis are genotypes GC 1-1, EsD 1-1, AcP bc.
PubMed ID
20480820 View in PubMed
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[Clinical analysis of 130 cases of Siberian silicosis]

https://arctichealth.org/en/permalink/ahliterature69571
Source
Nippon Igaku Hoshasen Gakkai Zasshi. 1995 Feb;55(3):121-8
Publication Type
Article
Date
Feb-1995
Author
A. Goto
S. Nawata
Author Affiliation
Department of Radiological Science, Tokyo Metropolitan College of Allied Medical Sciences.
Source
Nippon Igaku Hoshasen Gakkai Zasshi. 1995 Feb;55(3):121-8
Date
Feb-1995
Language
Japanese
Publication Type
Article
Keywords
Adult
Aged
Dust
English Abstract
Humans
Lung - radiography
Male
Middle Aged
Mining
Prisoners
Siberia
Silicosis - etiology - radiography
Tomography, X-Ray Computed
War
Abstract
One hundred thirty patients with Siberian silicosis occurring under exposure to large amounts of dust at labor in a siberian mine were analyzed for 67 clinical and 122 laboratory data. Eighty-eight of 130 patients are now alive, but 42 have already died. When they started work, 122 of the patients were under thirty years of age. The duration of work was 7 to 12 months for 17 patients, 13 to 18 for 43, and 19 to 24 for 40. Seventy-six of 99 patients were initially diagnosed with lung tuberculosis and 23 with silicosis. Almost all patients have complained of respiratory symptoms such as shortness of breath, cough, sputum, and cyanosis. All of the pulmonary function tests including %VC, FEV1.0/FVC, V25/height, RV, TLC, and DLco showed abnormal values. The chest roentogenograms showed 3 of type 1, 22 of type 2, 55 of type 3, and 124 of type 4. Of 124 type-4, large opacities, 84 were type A, 28 B, and 12 C. Of 416 small opacities, 144 were type P, 191 Q, and 81 R. The complications and secondary changes that appeared with progression of the disease were lung emphysema, hilar and mediastinal lymphnode enlargement, egg shell calcification in lymphnode, and bulla or bleb.
PubMed ID
7731765 View in PubMed
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[Clinical and experimental studies of metabolic response to chronic exposure to coal dust].

https://arctichealth.org/en/permalink/ahliterature135148
Source
Med Tr Prom Ekol. 2011;(2):15-21
Publication Type
Article
Date
2011
Author
D V Fomenko
L G Gorokhova
N I Panev
A S Kazitskaia
O I Bondarev
Source
Med Tr Prom Ekol. 2011;(2):15-21
Date
2011
Language
Russian
Publication Type
Article
Keywords
Adult
Animals
Anthracosilicosis - blood - etiology - immunology - metabolism
Chronic Disease
Coal
Coal Mining
Dust
Humans
Immunity, Humoral - drug effects
Inhalation Exposure - adverse effects
Lipid Metabolism
Lipids - blood
Lung - drug effects - pathology
Male
Middle Aged
Occupational Exposure - adverse effects
Rats
Russia
Abstract
In miners anthracosilicosis is caused by chronic exposure to coal dust and is characterized by progressive development of the inflammatory process, the expressed disorders of lipid metabolism, and immunodeficiency. In the experiment we revealed the stages of anthracosilicosis development according to which adequate measures of prevention and correction of the disorders caused by long exposure of an organism to coal dust are recommended.
PubMed ID
21506373 View in PubMed
Less detail

[Coronary heart disease, its risk factors and somatic types in coal miners having chronic dust-related diseases of lungs].

https://arctichealth.org/en/permalink/ahliterature187431
Source
Med Tr Prom Ekol. 2002;(10):30-4
Publication Type
Article
Date
2002
Author
N G Stankevich
Ia A Gorbatovskii
S N Filimonov
N I Panev
M V Luk'ianova
Source
Med Tr Prom Ekol. 2002;(10):30-4
Date
2002
Language
Russian
Publication Type
Article
Keywords
Adult
Aged
Catchment Area (Health)
Chronic Disease
Coal Mining
Dust
Female
Humans
Lung Diseases - epidemiology - etiology
Male
Middle Aged
Myocardial Ischemia - epidemiology - etiology
Occupational Diseases - epidemiology
Risk factors
Russia - epidemiology
Somatoform Disorders - epidemiology
Abstract
Studies covered incidence of coronary heart disease, its risk factors and features of constitutional types among Kouzbass coal miners suffering from anthracosilicosis and chronic dust bronchitis. Findings are reliably higher incidence of coronary heart disease among coal miners having lung diseases caused by dust. Coronary heart disease among the miners with anthracosilicosis is favored by arterial hypertension, overweight and hypersthenic constitutional type, that among those with dust bronchitis is favored only by overweight.
PubMed ID
12474281 View in PubMed
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30 records – page 1 of 3.