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[Chronic occupational bronchitis in workers of coal extracting enterprises in Kouzbass: role of endogenous factors].

https://arctichealth.org/en/permalink/ahliterature143489
Source
Med Tr Prom Ekol. 2010;(3):37-40
Publication Type
Article
Date
2010
Author
N I Gafarov
V V Zakharenkov
N I Panev
A V Burdein
V P Puzyrev
A A Rudko
Source
Med Tr Prom Ekol. 2010;(3):37-40
Date
2010
Language
Russian
Publication Type
Article
Keywords
Acid Phosphatase - blood
Adult
Biological Markers - blood
Bronchitis, Chronic - blood - epidemiology - genetics
Carboxylesterase - blood
Coal
Coal Mining
Dust
Genetic Predisposition to Disease
Genotype
Haptoglobins - metabolism
Humans
Middle Aged
Occupational Diseases - blood - epidemiology - genetics
Occupational Exposure - adverse effects
Polymorphism, Genetic
Prevalence
Siberia - epidemiology
Vitamin D-Binding Protein - blood
Abstract
The authors studied distribution of biochemical markers for HP, GC, EsD, AcP genes, polymorphism of GSTT1 (GST-theta 1), GSTM1 (GST-mu 1), locus WNTR of NOS3 gene (alleles A/B) in chronic dust bronchitis patients and in apparently healthy individuals. Genotypes EsD 1-2 and AcP bb individuals were proved to be most prone to the disease. Endogenous resistent factors for chronic dust bronchitis are genotypes GC 1-1, EsD 1-1, AcP bc.
PubMed ID
20480820 View in PubMed
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[Clinical and experimental studies of metabolic response to chronic exposure to coal dust].

https://arctichealth.org/en/permalink/ahliterature135148
Source
Med Tr Prom Ekol. 2011;(2):15-21
Publication Type
Article
Date
2011
Author
D V Fomenko
L G Gorokhova
N I Panev
A S Kazitskaia
O I Bondarev
Source
Med Tr Prom Ekol. 2011;(2):15-21
Date
2011
Language
Russian
Publication Type
Article
Keywords
Adult
Animals
Anthracosilicosis - blood - etiology - immunology - metabolism
Chronic Disease
Coal
Coal Mining
Dust
Humans
Immunity, Humoral - drug effects
Inhalation Exposure - adverse effects
Lipid Metabolism
Lipids - blood
Lung - drug effects - pathology
Male
Middle Aged
Occupational Exposure - adverse effects
Rats
Russia
Abstract
In miners anthracosilicosis is caused by chronic exposure to coal dust and is characterized by progressive development of the inflammatory process, the expressed disorders of lipid metabolism, and immunodeficiency. In the experiment we revealed the stages of anthracosilicosis development according to which adequate measures of prevention and correction of the disorders caused by long exposure of an organism to coal dust are recommended.
PubMed ID
21506373 View in PubMed
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[Coronary heart disease, its risk factors and somatic types in coal miners having chronic dust-related diseases of lungs].

https://arctichealth.org/en/permalink/ahliterature187431
Source
Med Tr Prom Ekol. 2002;(10):30-4
Publication Type
Article
Date
2002
Author
N G Stankevich
Ia A Gorbatovskii
S N Filimonov
N I Panev
M V Luk'ianova
Source
Med Tr Prom Ekol. 2002;(10):30-4
Date
2002
Language
Russian
Publication Type
Article
Keywords
Adult
Aged
Catchment Area (Health)
Chronic Disease
Coal Mining
Dust
Female
Humans
Lung Diseases - epidemiology - etiology
Male
Middle Aged
Myocardial Ischemia - epidemiology - etiology
Occupational Diseases - epidemiology
Risk factors
Russia - epidemiology
Somatoform Disorders - epidemiology
Abstract
Studies covered incidence of coronary heart disease, its risk factors and features of constitutional types among Kouzbass coal miners suffering from anthracosilicosis and chronic dust bronchitis. Findings are reliably higher incidence of coronary heart disease among coal miners having lung diseases caused by dust. Coronary heart disease among the miners with anthracosilicosis is favored by arterial hypertension, overweight and hypersthenic constitutional type, that among those with dust bronchitis is favored only by overweight.
PubMed ID
12474281 View in PubMed
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[Hemostasis disorders in coal miners].

https://arctichealth.org/en/permalink/ahliterature147613
Source
Med Tr Prom Ekol. 2009;(9):22-5
Publication Type
Article
Date
2009
Author
S N Filimonov
V V Zakharenkov
N I Panev
A V Burdein
L A Danilevskaia
N N Epifantseva
Source
Med Tr Prom Ekol. 2009;(9):22-5
Date
2009
Language
Russian
Publication Type
Article
Keywords
Adult
Blood Coagulation Disorders - blood - epidemiology - etiology
Coal Mining
Hemostasis - physiology
Humans
Incidence
Middle Aged
Occupational Diseases - blood - epidemiology - etiology
Occupational Exposure - adverse effects
Russia - epidemiology
Abstract
Findings are that coal miners having long contact with vibration instruments and coal dust develop endothelial dysfunction, increased platelets aggregation, hypercoagulation and lower anticoagulation activity. The hemostasis disorders revealed could result in earlier coronary atherosclerosis development in workers exposed to vibration.
PubMed ID
19882776 View in PubMed
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[The role of genetic factors in the development of chronic dust bronchitis in workers of coal mining enterprises of Kuzbass].

https://arctichealth.org/en/permalink/ahliterature105655
Source
Gig Sanit. 2013 Jul-Aug;(4):44-7
Publication Type
Article
Author
N I Gafarov
V V Zakharenkov
N I Panev
A N Kucher
M B Freidin
A A Rudko
Source
Gig Sanit. 2013 Jul-Aug;(4):44-7
Language
Russian
Publication Type
Article
Keywords
Adult
Air Pollutants, Occupational - toxicity
Bronchitis, Chronic - chemically induced - genetics
Case-Control Studies
Coal Mining
Dust
Gene-Environment Interaction
Genetic Predisposition to Disease
Genotype
Glutathione Transferase - genetics
Humans
Male
Middle Aged
Nitric Oxide Synthase Type III - genetics
Occupational Diseases - chemically induced - genetics
Polymorphism, Genetic
Siberia
Abstract
The distribution of genotypes of HP, GC, EsD, AsP and polymorphisms GSTT1 (GST-theta1) and GSTM1 (GST-micro1) and NOS3 (polymorphism VNTR4) in miners with chronic dust bronchitis, and in those without this occupational disease has been studied The carriers of genotypes of genotypes EsD 1-2, AsP bb were shown to be more prone to develop chronic dust bronchitis. Endogenous factors of resistance to the disease are the genotypes GC 1-1, EsD 1-1, AsP bc.
PubMed ID
24340601 View in PubMed
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