Skip header and navigation

1 records – page 1 of 1.

Gluten contamination in the Canadian commercial oat supply.
Food Addit Contam Part A Chem Anal Control Expo Risk Assess. 2011 Jun;28(6):705-10
Publication Type
T B Koerner
C. Cléroux
C. Poirier
I. Cantin
A. Alimkulov
H. Elamparo
Author Affiliation
Bureau of Chemical Safety, Food Directorate, Health Canada, Ottawa, ON, Canada.
Food Addit Contam Part A Chem Anal Control Expo Risk Assess. 2011 Jun;28(6):705-10
Publication Type
Avena sativa - chemistry
Celiac Disease - diet therapy
Cereals - chemistry - economics - standards
Diet, Gluten-Free - economics - standards
Enzyme-Linked Immunosorbent Assay
Food Contamination
Food Hypersensitivity - diet therapy
Food Labeling - standards
Food, Organic - analysis - economics - standards
Glutens - adverse effects - analysis
Legislation, Food
Limit of Detection
Seeds - chemistry
A growing body of evidence suggests that a majority of people with celiac disease and on a gluten-free diet can safely consume pure oats in moderate amounts; however, previous studies have indicated that the commercial oat supply in other countries, and in Canada to some extent, is contaminated with other grains. This study has confirmed that the commercial oat supply in Canada is heavily contaminated with gluten from other grains. Approximately 88% of the oat samples (n = 133) were contaminated above 20 mg kg(-1) and there were no differences between the oat types tested. Only one gluten-free variety of oats was analysed and it consistently provided negative results in all analyses. It is difficult to determine where the contamination originates, but there are possibilities for cross-contamination in the field, in the transport of the grain, in the storage of the grain, and in the milling and packaging facilities. It is clear from this study that only those products that have been certified 'pure' oats would be appropriate for a gluten-free diet.
Cites: N Engl J Med. 2002 Jan 17;346(3):180-811796853
Cites: J Allergy Clin Immunol. 2010 Feb;125(2 Suppl 2):S116-2520042231
Cites: N Engl J Med. 2004 Nov 4;351(19):2021-215525734
Cites: Allergy. 2005 Jun;60(6):815-2115876313
Cites: J Hum Nutr Diet. 2005 Jun;18(3):163-915882378
Cites: Aliment Pharmacol Ther. 2006 Mar 1;23(5):559-7516480395
Cites: J Am Diet Assoc. 2006 May;106(5):665; discussion 665-616647318
Cites: Postgrad Med J. 2006 Oct;82(972):672-817068278
Cites: Can J Gastroenterol. 2007 Oct;21(10):649-5117948135
Cites: Scand J Gastroenterol. 2007 Nov;42(11):1302-517852883
Cites: Eur J Gastroenterol Hepatol. 2008 Jun;20(6):545-5418467914
Cites: Curr Opin Gastroenterol. 2008 Nov;24(6):687-9119122516
Cites: Adv Food Nutr Res. 2009;57:235-8519595389
Cites: Gastroenterol Clin North Am. 2009 Sep;38(3):433-4619699406
Cites: Ann Allergy Asthma Immunol. 2010 Jan;104(1):60-520143647
Cites: Curr Opin Gastroenterol. 2010 Mar;26(2):116-2220040864
Cites: J Am Diet Assoc. 2003 Mar;103(3):376-912616264
PubMed ID
21623493 View in PubMed
Less detail