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Associations among 25-year trends in diet, cholesterol and BMI from 140,000 observations in men and women in Northern Sweden.
Nutr J. 2012;11:40
Publication Type
Ingegerd Johansson
Lena Maria Nilsson
Birgitta Stegmayr
Kurt Boman
Göran Hallmans
Anna Winkvist
Author Affiliation
Department of Odontology, Umeå University, 901 87, Umeå, Sweden.
Nutr J. 2012;11:40
Publication Type
Alcohol Drinking - adverse effects - trends
Body mass index
Cholesterol - blood
Cohort Studies
Cross-Sectional Studies
Diet Surveys
Diet, Carbohydrate-Restricted - adverse effects
Diet, High-Fat - adverse effects
Diet, Reducing - adverse effects - trends
Follow-Up Studies
Health promotion
Mass Media - trends
Middle Aged
Patient Compliance - ethnology
Sex Characteristics
Weight Gain
In the 1970s, men in northern Sweden had among the highest prevalences of cardiovascular diseases (CVD) worldwide. An intervention program combining population- and individual-oriented activities was initiated in 1985. Concurrently, collection of information on medical risk factors, lifestyle and anthropometry started. Today, these data make up one of the largest databases in the world on diet intake in a population-based sample, both in terms of sample size and follow-up period. The study examines trends in food and nutrient intake, serum cholesterol and body mass index (BMI) from 1986 to 2010 in northern Sweden.
Cross-sectional information on self-reported food and nutrient intake and measured body weight, height, and serum cholesterol were compiled for over 140,000 observations. Trends and trend breaks over the 25-year period were evaluated for energy-providing nutrients, foods contributing to fat intake, serum cholesterol and BMI.
Reported intake of fat exhibited two significant trend breaks in both sexes: a decrease between 1986 and 1992 and an increase from 2002 (women) or 2004 (men). A reverse trend was noted for carbohydrates, whereas protein intake remained unchanged during the 25-year period. Significant trend breaks in intake of foods contributing to total fat intake were seen. Reported intake of wine increased sharply for both sexes (more so for women) and export beer increased for men. BMI increased continuously for both sexes, whereas serum cholesterol levels decreased during 1986 - 2004, remained unchanged until 2007 and then began to rise. The increase in serum cholesterol coincided with the increase in fat intake, especially with intake of saturated fat and fats for spreading on bread and cooking.
Men and women in northern Sweden decreased their reported fat intake in the first 7 years (1986-1992) of an intervention program. After 2004 fat intake increased sharply for both genders, which coincided with introduction of a positive media support for low carbohydrate-high-fat (LCHF) diet. The decrease and following increase in cholesterol levels occurred simultaneously with the time trends in food selection, whereas a constant increase in BMI remained unaltered. These changes in risk factors may have important effects on primary and secondary prevention of cardiovascular disease (CVD).
Cites: Scand J Public Health Suppl. 2003;61:18-2414660243
Cites: J Intern Med. 2007 Apr;261(4):366-7417391111
Cites: Scand J Public Health Suppl. 2003;61:31-714660245
Cites: Int J Epidemiol. 1985 Jun;14(2):285-924018996
Cites: Hum Nutr Clin Nutr. 1985;39 Suppl 1:5-414044297
Cites: Am J Epidemiol. 1986 Dec;124(6):903-153776973
Cites: Stat Med. 2000 Feb 15;19(3):335-5110649300
Cites: Eur J Clin Nutr. 2007 May;61(5):575-8117136037
Cites: N Engl J Med. 2009 Feb 26;360(9):859-7319246357
Cites: Arch Intern Med. 2009 Apr 13;169(7):659-6919364995
Cites: Eur Heart J. 2009 May;30(9):1046-5619141562
Cites: Public Health Nutr. 2009 Sep;12(9):1477-8419144238
Cites: Eur Urol. 2009 Jan;55(1):249-5020050018
Cites: Int J Epidemiol. 2010 Apr;39(2):504-1819959603
Cites: Public Health Nutr. 2010 Jun;13(6A):932-820513263
Cites: Eur J Clin Nutr. 2010 Aug;64(8):905-1320502473
Cites: Ann Intern Med. 2010 Sep 7;153(5):289-9820820038
Cites: Cancer Causes Control. 2010 Oct;21(10):1533-4420512657
Cites: Am J Clin Nutr. 2010 Oct;92(4):967-7420826627
Cites: Diabetes Care. 2010 Dec;33(12):2684-9120693352
Cites: J Intern Med. 2011 Feb;269(2):219-3121158982
Cites: Environ Health. 2011;10:3321504558
Cites: Acta Neurol Scand. 2012 Jun;125(6):382-821793808
Cites: Lancet. 2000 Feb 26;355(9205):675-8710703799
Cites: J Intern Med. 2000 May;247(5):579-8710809997
Cites: Public Health Nutr. 2001 Aug;4(4):919-2711527517
Cites: Scand J Public Health Suppl. 2001;56:13-2011681559
Cites: Public Health Nutr. 2002 Jun;5(3):487-9612003662
Cites: Lancet. 2003 May 3;361(9368):1496-50112737858
Cites: Scand J Public Health Suppl. 2003;61:9-1714660242
Cites: World Health Stat Q. 1987;40(2):171-843617777
Cites: Prog Med Chem. 1988;25:291-3383076969
Cites: Int J Epidemiol. 1993 Dec;22(6):1026-378144283
Cites: Stroke. 1994 Sep;25(9):1738-458073452
Cites: Lancet. 1994 Nov 19;344(8934):1383-97968073
Cites: BMJ. 1995 Oct 14;311(7011):986-97580640
Cites: Int J Epidemiol. 1997;26 Suppl 1:S6-149126529
Cites: J Intern Med. 1998 Feb;243(2):99-1079566637
Cites: Scand J Prim Health Care. 1998 Sep;16(3):171-69800231
Cites: Circulation. 1999 Feb 16;99(6):779-859989963
Cites: Scand J Public Health. 2005;33(4):321-4; discussion 24316087495
Cites: Lancet. 2005 Oct 8;366(9493):1267-7816214597
Cites: Arch Intern Med. 2006 Feb 13;166(3):285-9316476868
Cites: Gut. 2006 Oct;55(10):1461-616638790
Cites: N Engl J Med. 2006 Nov 9;355(19):1991-200217093250
Cites: J Intern Med. 2006 Dec;260(6):551-917116006
Cites: Nat Clin Pract Cardiovasc Med. 2007 Jan;4(1):34-4117180148
Cites: Scand J Public Health Suppl. 2003;61:25-3014660244
PubMed ID
22686621 View in PubMed
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