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An outbreak of Listeria monocytogenes serotype 3a infections from butter in Finland.

https://arctichealth.org/en/permalink/ahliterature198523
Source
J Infect Dis. 2000 May;181(5):1838-41
Publication Type
Article
Date
May-2000
Author
O. Lyytikäinen
T. Autio
R. Maijala
P. Ruutu
T. Honkanen-Buzalski
M. Miettinen
M. Hatakka
J. Mikkola
V J Anttila
T. Johansson
L. Rantala
T. Aalto
H. Korkeala
A. Siitonen
Author Affiliation
Department of Infectious Disease Epidemiology, National Public Health Institute, FIN-00300 Helsinki, Finland. outi.lyytikainen@ktl. fi.
Source
J Infect Dis. 2000 May;181(5):1838-41
Date
May-2000
Language
English
Publication Type
Article
Keywords
Adolescent
Adult
Aged
Aged, 80 and over
Butter - microbiology
Case-Control Studies
Child
Cross Infection - epidemiology
Dairying
Disease Outbreaks
Female
Finland - epidemiology
Humans
Incidence
Listeria monocytogenes - classification
Listeriosis - epidemiology - etiology - transmission
Male
Middle Aged
Serotyping
Abstract
In February 1999, an outbreak of listeriosis caused by Listeria monocytogenes serotype 3a occurred in Finland. All isolates were identical. The outbreak strain was first isolated in 1997 in dairy butter. This dairy began delivery to a tertiary care hospital (TCH) in June 1998. From June 1998 to April 1999, 25 case patients were identified (20 with sepsis, 4 with meningitis, and 1 with abscess; 6 patients died). Patients with the outbreak strain were more likely to have been admitted to the TCH than were patients with other strains of L. monocytogenes (60% vs. 8%; odds ratio, 17.3; 95% confidence interval, 2.8-136.8). Case patients admitted to the TCH had been hospitalized longer before cultures tested positive than had matched controls (median, 31 vs. 10 days; P=.008). An investigation found the outbreak strain in packaged butter served at the TCH and at the source dairy. Recall of the product ended the outbreak.
PubMed ID
10823797 View in PubMed
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Source
Duodecim. 2000;116(19):2111-8
Publication Type
Article
Date
2000
Author
O. Lyytikäinen
A. Siitonen
T. Johansson
S. Lukinmaa
J. Mikkola
P. Ruutu
Author Affiliation
Kansanterveyslaitos, infektioepidemiologian osasto Mannerheimintie 166, 00300 Helsinki. outi.lyytikainen@ktl.fi
Source
Duodecim. 2000;116(19):2111-8
Date
2000
Language
Finnish
Publication Type
Article
Keywords
Finland - epidemiology
Food Microbiology
Foodborne Diseases - microbiology - prevention & control
Humans
Listeriosis - diagnosis - epidemiology - therapy
Risk factors
PubMed ID
12017732 View in PubMed
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Reactive arthritis after an outbreak of Yersinia pseudotuberculosis serotype O:3 infection.

https://arctichealth.org/en/permalink/ahliterature184063
Source
Ann Rheum Dis. 2003 Sep;62(9):866-9
Publication Type
Article
Date
Sep-2003
Author
T. Hannu
L. Mattila
J P Nuorti
P. Ruutu
J. Mikkola
A. Siitonen
M. Leirisalo-Repo
Author Affiliation
Department of Medicine, Division of Rheumatology, Helsinki University Central Hospital, Finland.
Source
Ann Rheum Dis. 2003 Sep;62(9):866-9
Date
Sep-2003
Language
English
Publication Type
Article
Keywords
Adolescent
Adult
Aged
Arthritis, Reactive - diagnosis - epidemiology
Child
Child, Preschool
Disease Outbreaks
Female
Finland - epidemiology
Health Surveys
Humans
Male
Middle Aged
Yersinia pseudotuberculosis Infections - diagnosis - epidemiology
Abstract
To determine the occurrence and clinical characteristics of reactive arthritis (ReA) after an outbreak of Yersinia pseudotuberculosis serotype O:3 infection.
From 15 October to 6 November 1998, a widespread outbreak of Y pseudotuberculosis serotype O:3 occurred in Finland. A questionnaire on musculoskeletal symptoms was mailed to 38 patients with infection confirmed by culture. All patients who reported joint symptoms were interviewed by phone and their medical records of outpatient visits or hospital admission because of recent joint symptoms were reviewed.
Thirty three of 38 (87%) patients returned the questionnaire. Reactive musculoskeletal symptoms were reported by 5/33 (15%): four patients (12%) fulfilled the criteria for ReA and one additional patient had reactive enthesopathy. The patients with ReA were adults (age range 40-47 years), whereas the patient with reactive enthesopathy was a 14 year old boy. In all patients with ReA, the arthritis was polyarticular. In addition to peripheral arthritis, other musculoskeletal symptoms included sacroiliitis (one patient), pain in Achilles tendon (one patient), and heel pain (two patients). HLA-B27 was positive in all the three patients tested. In three of four patients with ReA, the duration of acute arthritis was over six months.
Y pseudotuberculosis serotype O:3 infection is frequently associated with ReA and the clinical picture is severe.
Notes
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PubMed ID
12922960 View in PubMed
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Seasonal increase in human IgE and IgG4 antisaliva antibodies to Aedes mosquito bites.

https://arctichealth.org/en/permalink/ahliterature207001
Source
Int Arch Allergy Immunol. 1997 Dec;114(4):367-72
Publication Type
Article
Date
Dec-1997
Author
K. Palosuo
H. Brummer-Korvenkontio
J. Mikkola
T. Sahi
T. Reunala
Author Affiliation
Department of Dermatology, University Hospital and University of Helsinki, Finland.
Source
Int Arch Allergy Immunol. 1997 Dec;114(4):367-72
Date
Dec-1997
Language
English
Publication Type
Article
Keywords
Adult
Aedes - immunology
Animals
Female
Finland
Humans
Hypersensitivity - immunology
Immunoglobulin E - blood
Immunoglobulin G - blood
Immunoglobulins - blood
Insect Bites and Stings - immunology
Male
Middle Aged
Saliva - immunology
Salivary Proteins and Peptides - immunology
Seasons
Abstract
Mosquito bite-sensitive subjects frequently have circulating IgE and IgG4 antibodies to Aedes mosquito saliva proteins.
In the present study we examined the antibody response during a mosquito season in 14 subjects living in Finnish Lapland. Immunoblotting was performed with Aedes communis saliva and the 22- and 36-kD antisaliva antibody bands were analyzed.
The preseason sera showed IgE antibodies to the main saliva antigens in 12, IgG4 antibodies in all 14 and IgG1 antibodies in 12 subjects, and the postseason sera in all but 1 subject. The postseason sera showed significantly more intense IgE (p
PubMed ID
9414141 View in PubMed
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