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Genetic markers for chronic bronchitis and peak expiratory flow in the Copenhagen Male Study.

https://arctichealth.org/en/permalink/ahliterature67783
Source
Dan Med Bull. 1993 Jun;40(3):378-80
Publication Type
Article
Date
Jun-1993
Author
J. Vestbo
H O Hein
P. Suadicani
H. Sørensen
F. Gyntelberg
Author Affiliation
Department of Occupational Medicine, Rigshospitalet, Copenhagen.
Source
Dan Med Bull. 1993 Jun;40(3):378-80
Date
Jun-1993
Language
English
Publication Type
Article
Keywords
ABO Blood-Group System
Aged
Blood Group Antigens
Bronchitis - blood - epidemiology - physiopathology
Chronic Disease
Complement C3 - physiology
Cross-Sectional Studies
Denmark - epidemiology
Follow-Up Studies
Genetic markers
Humans
Male
Middle Aged
Peak Expiratory Flow Rate
Prevalence
Research Support, Non-U.S. Gov't
Smoking - adverse effects
Abstract
The associations between four major blood groups, ABH secretor status, and complement C3, and chronic bronchitis and peak expiratory flow were examined in 3387 men, aged 55-74 years. Presence of chronic bronchitis was assessed using the British Medical Research Council (BMRC) questionnaire. Men with NS- in the MNS system had significantly less chronic bronchitis than others, i.e. 11.4% versus 16.0% (p
PubMed ID
8339607 View in PubMed
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Genetic markers for peptic ulcer. A study of 3387 men aged 54 to 74 years: the Copenhagen Male Study.

https://arctichealth.org/en/permalink/ahliterature210276
Source
Scand J Gastroenterol. 1997 Jan;32(1):16-21
Publication Type
Article
Date
Jan-1997
Author
H O Hein
P. Suadicani
F. Gyntelberg
Author Affiliation
Epidemiological Research Unit, Copenhagen University Hospital, Denmark.
Source
Scand J Gastroenterol. 1997 Jan;32(1):16-21
Date
Jan-1997
Language
English
Publication Type
Article
Keywords
ABO Blood-Group System - genetics
Aged
Chi-Square Distribution
Genetic markers
Humans
Lewis Blood-Group System - genetics
Male
Middle Aged
Peptic Ulcer - blood - epidemiology - genetics
Phenotype
Prevalence
Scandinavia - epidemiology
Selection Bias
Abstract
Knowledge on the genetic risk of peptic ulcer has predominantly been based on hospital materials. To minimize selection bias, we tested the association between some genetic markers and the risk of peptic ulcer in a large-scale epidemiologic design.
Some 3387 white men aged 55-74 years were investigated and reported their history of peptic ulcer. Information about hospitalization and operation was collected from registers.
The lifetime prevalence of peptic ulcer in men with the Lewis phenotype Le(a + b-) and non-secretors of ABH antigen was 15%, significantly higher than others, 11% (P
Notes
Comment In: Scand J Gastroenterol. 1999 Feb;34(2):219-2010192205
PubMed ID
9018761 View in PubMed
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[Genetic markers for stomach ulcer. A study of 3,387 men aged 54-74 years from The Copenhagen Male Study].

https://arctichealth.org/en/permalink/ahliterature204573
Source
Ugeskr Laeger. 1998 Aug 24;160(35):5045-9
Publication Type
Article
Date
Aug-24-1998
Author
H O Hein
P. Suadicani
F. Gyntelberg
Author Affiliation
H:S Bispebjerg Hospital, epidemiologisk forskningsenhed, arbejds- og miljømedicinsk klinik.
Source
Ugeskr Laeger. 1998 Aug 24;160(35):5045-9
Date
Aug-24-1998
Language
Danish
Publication Type
Article
Keywords
Aged
Blood Grouping and Crossmatching
Cohort Studies
Denmark - epidemiology
Disease Susceptibility
Duodenal Ulcer - epidemiology - etiology - genetics
Genetic markers
Helicobacter Infections - complications
Humans
Male
Middle Aged
Phenotype
Prevalence
Registries
Risk factors
Stomach Ulcer - epidemiology - etiology - genetics
Abstract
Knowledge on the genetic risk of peptic ulcer has predominantly been based on hospital materials. To minimize selection bias, we tested the association between some genetic markers and the risk of peptic ulcer in a large-scale epidemiologic design.
Some 3,387 white men aged 54-74 years were investigated and reported their history of peptic ulcer. Information about hospitalization and operation was collected from registers.
The lifetime prevalence of peptic ulcer in men with Lewis phenotype Le(a + b-) and non-secretors of ABH antigen was 15%, significantly higher than others, 11%, p; the risk in phenotypes O and A were equally high, 12%, and among other ABO phenotypes, 7%, p
PubMed ID
9739607 View in PubMed
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The Lewis blood group--a new genetic marker of obesity.

https://arctichealth.org/en/permalink/ahliterature175242
Source
Int J Obes (Lond). 2005 May;29(5):540-2
Publication Type
Article
Date
May-2005
Author
H O Hein
P. Suadicani
F. Gyntelberg
Author Affiliation
Epidemiological Research Unit, Clinic of Environmental and Occupational Medicine, Bispebjerg University Hospital, Copenhagen NV, Denmark. hoh01@bbh.hosp.dk
Source
Int J Obes (Lond). 2005 May;29(5):540-2
Date
May-2005
Language
English
Publication Type
Article
Keywords
Aged
Body mass index
Cross-Sectional Studies
Denmark - epidemiology
Genetic markers
Humans
Lewis Blood-Group System - genetics
Male
Middle Aged
Obesity - epidemiology - genetics
Phenotype
Prevalence
Abstract
In a cross-sectional epidemiological study including 3290 men aged 53-75 y, mean=63, we tested the hypothesis that the Lewis phenotype Le(a-b-) is a genetic marker of obesity. All men were Lewis typed and measurements were made of height, weight, neck circumference, and hip fat fold. Obesity was defined as a body mass index >/=30 kg/m(2). Totally 291 men (8.8%) were obese; 9.6% of the men had the Le(a-b-) phenotype. Le(a-b-) men had a higher prevalence of obesity than others, 15.6 vs 8.1%, odds ratio (95% confidence interval): 2.1(1.5-2.9), P
PubMed ID
15832169 View in PubMed
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Weight changes and risk of ischaemic heart disease for middle aged and elderly men. An 8-year follow-up in the Copenhagen Male study.

https://arctichealth.org/en/permalink/ahliterature54553
Source
J Cardiovasc Risk. 1997 Feb;4(1):25-32
Publication Type
Article
Date
Feb-1997
Author
P. Suadicani
H O Hein
F. Gyntelberg
Author Affiliation
Copenhagen Male Study, Rigshospitalet, Denmark.
Source
J Cardiovasc Risk. 1997 Feb;4(1):25-32
Date
Feb-1997
Language
English
Publication Type
Article
Keywords
Cohort Studies
Denmark - epidemiology
Follow-Up Studies
Genetic markers
Humans
Incidence
Lewis Blood-Group System - genetics
Life Style
Male
Middle Aged
Myocardial Ischemia - epidemiology
Phenotype
Prospective Studies
Research Support, Non-U.S. Gov't
Risk factors
Time Factors
Weight Gain
Weight Loss
Abstract
BACKGROUND: Weight gain and weight loss are determined by a complex interplay of health, lifestyle and genetic factors. There is controversy concerning whether weight changes are associated with an increased risk of ischaemic heart disease (IHD). METHODS: The Copenhageri Male Study was initiated in 1970/1971. This paper presents the results of a prospective study using a baseline comprising survivors examined 15 years later in 1985/1986 including 2903 men (aged 53-74 years) without cardiovascular disease. They were classified according to their weight change from 1970/1971 to 1985/1986. RESULTS: There were no differences among the incidences of IHD during the period 1985/1986-1993 for men who had lost weight (> 5% weight loss), men whose weight had remained constant (within +/-5%) and men who had gained weight (> 5% weight gain); the incidence rates were 7.8, 8.4 and 7.6%, respectively, NS. Adjustment for age and confounders including disease history had no influence on this result. Only for men who had already been overweight at the time of initial study in 1970/1971 (body mass index > 28 kg/m2) was there a slightly increased risk of IHD among those who gained weight. Men in this group who gained weight were characterized by a significantly (P
PubMed ID
9215517 View in PubMed
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