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Adherence to a healthy Nordic food index and breast cancer risk: results from a Swedish cohort study.

https://arctichealth.org/en/permalink/ahliterature271574
Source
Cancer Causes Control. 2015 Jun;26(6):893-902
Publication Type
Article
Date
Jun-2015
Author
Yingjun Li
Nina Roswall
Sven Sandin
Peter Ström
Hans-Olov Adami
Elisabete Weiderpass
Source
Cancer Causes Control. 2015 Jun;26(6):893-902
Date
Jun-2015
Language
English
Publication Type
Article
Keywords
Adult
Breast Neoplasms - epidemiology
Cohort Studies
Diet
Female
Health Surveys
Humans
Incidence
Middle Aged
Premenopause
Prospective Studies
Risk
Sweden - epidemiology
Abstract
A healthy Nordic dietary pattern has shown beneficial effects in relation to several chronic diseases. However, no study has evaluated the association between a healthy Nordic food index (HNFI) and risk of breast cancer.
We conducted a prospective cohort study including 44,296 women, aged 29-49 at baseline in 1991-1992, who completed a food frequency questionnaire at baseline, and have been followed up ever since, through the Swedish Cancer Registry and Cause of Death Registry. Each woman was assigned a HNFI score ranging from 0 to 6. We calculated multivariable relative risks (RRs) and 95% confidence intervals (CIs) using Poisson regression models with attained age as the underlying timescale. The association between the HNFI and risk of breast cancer was assessed both overall, by menopausal status and by hormone receptor status.
A total of 1,464 breast cancer cases were diagnosed during a median follow-up time of 20 years. A higher adherence to the HNFI was not associated with a lower risk of breast cancer overall, nor of varied hormone receptor status, or when we examining premenopausal and postmenopausal women separately. The multivariable RRs (95% CI) for breast cancer per 1-point increment in the HNFI were 1.02 (95% CI 0.98-1.06) for all women, 1.01 (95% CI 0.95-1.08) for premenopausal women, and 1.02 (95% CI 0.97-1.07) for postmenopausal women.
Adherence to a HNFI was not associated with breast cancer incidence in this cohort of relatively young women, regardless of menopausal status or hormone receptor status.
PubMed ID
25783459 View in PubMed
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Adherence to the healthy Nordic food index and total and cause-specific mortality among Swedish women.

https://arctichealth.org/en/permalink/ahliterature268918
Source
Eur J Epidemiol. 2015 Jun;30(6):509-17
Publication Type
Article
Date
Jun-2015
Author
Nina Roswall
Sven Sandin
Marie Löf
Guri Skeie
Anja Olsen
Hans-Olov Adami
Elisabete Weiderpass
Source
Eur J Epidemiol. 2015 Jun;30(6):509-17
Date
Jun-2015
Language
English
Publication Type
Article
Keywords
Adult
Cardiovascular Diseases - mortality
Cause of Death
Diet
Edible Grain
Female
Food Habits
Fruit
Health promotion
Humans
Middle Aged
Mortality
Neoplasms - mortality
Norway
Prospective Studies
Regression Analysis
Sweden - epidemiology
Vegetables
Abstract
Several healthy dietary patterns have been linked to longevity. Recently, a Nordic dietary pattern was associated with a lower overall mortality. No study has, however, investigated this dietary pattern in relation to cause-specific mortality. The aim of the present study was to examine the association between adherence to a healthy Nordic food index (consisting of wholegrain bread, oatmeal, apples/pears, root vegetables, cabbages and fish/shellfish) and overall mortality, and death by cardiovascular disease, cancer, injuries/suicide and other causes. We conducted a prospective analysis in the Swedish Women's Lifestyle and Health cohort, including 44,961 women, aged 29-49 years, who completed a food frequency questionnaire between 1991-1992, and have been followed up for mortality ever since, through Swedish registries. The median follow-up time is 21.3 years, and mortality rate ratios (MRR) were calculated using Cox Proportional Hazards Models. Compared to women with the lowest index score (0-1 points), those with the highest score (4-6 points) had an 18% lower overall mortality (MRR 0.82; 0.71-0.93, p
PubMed ID
25784368 View in PubMed
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Alcohol consumption, body mass index and breast cancer risk by hormone receptor status: Women' Lifestyle and Health Study.

https://arctichealth.org/en/permalink/ahliterature273881
Source
BMC Cancer. 2015;15:881
Publication Type
Article
Date
2015
Author
Aesun Shin
Sven Sandin
Marie Lof
Karen L Margolis
Kyeezu Kim
Elisabeth Couto
Hans Olov Adami
Elisabete Weiderpass
Source
BMC Cancer. 2015;15:881
Date
2015
Language
English
Publication Type
Article
Keywords
Adult
Alcohol Drinking - adverse effects - pathology
Body mass index
Breast Neoplasms - epidemiology - genetics - pathology
Female
Humans
Life Style
Middle Aged
Postmenopause
Receptors, Estrogen - blood - genetics
Receptors, Progesterone - genetics
Risk factors
Sweden
Abstract
We aimed to estimate the effect of alcohol consumption on breast cancer risk and to test whether overweight and obesity modifies this association.
We included in the analysis 45,233 women enrolled in the Swedish Women's Lifestyle and Health study between 1991 and 1992. Participants were followed for occurrence of breast cancer and death until December 2009. Poisson regression models were used, and analyses were done for overall breast cancer and for estrogen receptor positive or negative (ER+, ER-) and progesterone receptor positive and negative (PR+, PR-) tumors separately.
A total of 1,385 breast cancer cases were ascertained during the follow-up period. Overall, we found no statistically significant association between alcohol intake and breast cancer risk after adjustment for confounding, with an estimated relative risk (RR) of 1.01 (95 % CI: 0.98-1.04) for an increment in alcohol consumption of 5 g/day. A statistically significant elevated breast cancer risk associated with higher alcohol consumption was found only among women with BMI =25 (RR 1.03, 95 % CI 1.0-1.05 per 5 g/day increase).
An increase in breast cancer risk with higher alcohol consumption was found for breast cancers in women with a BMI =25 kg/m(2).
Notes
Cites: Cancer Epidemiol Biomarkers Prev. 2012 Jul;21(7):1203-1222564867
Cites: Alcohol Alcohol. 2000 Sep-Oct;35(5):417-2311022013
Cites: Cancer Causes Control. 2001 Jan;12(1):47-5911227925
Cites: Cancer Epidemiol Biomarkers Prev. 2002 Nov;11(11):1375-8112433714
Cites: Br J Cancer. 2002 Nov 18;87(11):1234-4512439712
Cites: Cancer Epidemiol Biomarkers Prev. 2004 Mar;13(3):405-1115006916
Cites: Alcohol Clin Exp Res. 2004 May;28(5):780-515166654
Cites: N Engl J Med. 1987 May 7;316(19):1169-733574367
Cites: J Pharmacol Exp Ther. 1988 May;245(2):407-123367299
Cites: Prev Med. 1988 Nov;17(6):676-823072560
Cites: J Natl Cancer Inst. 1993 May 5;85(9):722-78478958
Cites: Proc Natl Acad Sci U S A. 1995 Apr 25;92(9):3650-77731959
Cites: JAMA. 1996 Dec 4;276(21):1747-518940324
Cites: JAMA. 1998 Feb 18;279(7):535-409480365
Cites: Alcohol Clin Exp Res. 1998 Aug;22(5):994-79726268
Cites: Alcohol Clin Exp Res. 1999 Jun;23(6):976-8210397281
Cites: Ann Epidemiol. 2006 Mar;16(3):230-4016230024
Cites: Cancer Causes Control. 2006 Aug;17(6):759-7016783604
Cites: Cancer Causes Control. 2007 May;18(4):361-7317364225
Cites: Int J Cancer. 2008 Apr 15;122(8):1832-4118067133
Cites: Ann Oncol. 2009 Mar;20(3):550-518765462
Cites: Lancet Oncol. 2009 Nov;10(11):1033-419891056
Cites: Anticancer Res. 2010 Jan;30(1):1-820150611
Cites: J Clin Oncol. 2010 Sep 10;28(26):4052-720644083
Cites: Eur J Epidemiol. 2011 Feb;26(2):81-9021267637
Cites: BMJ. 2011;342:d158421474525
Cites: Br J Cancer. 2011 Aug 23;105(5):709-2221772329
Cites: Br J Cancer. 2011 Dec 6;105 Suppl 2:S14-822158312
PubMed ID
26552431 View in PubMed
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Alcohol consumption over time and mortality in the Swedish Women's Lifestyle and Health cohort.

https://arctichealth.org/en/permalink/ahliterature287321
Source
BMJ Open. 2016 11 02;6(11):e012862
Publication Type
Article
Date
11-02-2016
Author
Idlir Licaj
Sven Sandin
Guri Skeie
Hans-Olov Adami
Nina Roswall
Elisabete Weiderpass
Source
BMJ Open. 2016 11 02;6(11):e012862
Date
11-02-2016
Language
English
Publication Type
Article
Keywords
Adult
Alcohol Drinking - mortality
Cardiovascular Diseases - mortality
Female
Humans
Life Style
Middle Aged
Multivariate Analysis
Myocardial Ischemia - mortality
Neoplasms - mortality
Proportional Hazards Models
Prospective Studies
Registries
Risk factors
Sweden - epidemiology
Abstract
Alcohol consumption is steadily increasing in high-income countries but the harm and possible net benefits of light-to-moderate drinking remain controversial. We prospectively investigated the association between time-varying alcohol consumption and overall and cause-specific mortality among middle-aged women.
Among 48 249 women at baseline (33 404 at follow-up) in the prospective Swedish Women's Lifestyle and Health cohort, age 30-49 years at baseline, we used repeated information on alcohol consumption and combined this method with multiple imputation in order to maximise the number of participants and deaths included in the analyses. Multivariable Cox regression models were used to calculate HRs for overall and cause-specific mortality.
During >900 000 person/years, a total of 2100 deaths were recorded through Swedish registries. The median alcohol consumption increased from 2.3 g/day in 1991/1992 (baseline) to 4.7 g/day in 2004 (follow-up). Compared with light drinkers (0.1-1.5 g/day), a null association was observed for all categories of alcohol consumption with the exception of never drinkers. The HR comparing never with light drinkers was 1.46 (95% CI 1.22 to 1.74). There was a statistically significant negative trend between increasing alcohol consumption and cardiovascular and ischaemic heart diseases mortality. The results were similar when women with prevalent conditions were excluded.
In conclusion, in a cohort of young women, light alcohol consumption was protective for cardiovascular and ischaemic heart disease mortality but not for cancer and overall mortality.
Notes
Cites: Addiction. 2013 Dec;108(12):2051-723297738
Cites: Alcohol Res Health. 2003;27(1):39-5115301399
Cites: Stat Med. 2010 Dec 10;29(28):2920-3120842622
Cites: Addiction. 2013 Dec;108(12):2058-924237894
Cites: Am J Epidemiol. 2014 May 1;179(9):1049-5924670372
Cites: BMJ Open. 2014 Jul 03;4(7):e00524524993766
Cites: Addiction. 1998 Feb;93(2):219-299624723
Cites: IARC Monogr Eval Carcinog Risks Hum. 2012;100(Pt E):1-53823193840
Cites: Ann Oncol. 2013 Feb;24(2):301-822910838
Cites: J Stud Alcohol. 1996 Sep;57(5):494-5068858547
Cites: J Womens Health (Larchmt). 2014 May;23(5):373-8124611563
Cites: N Engl J Med. 1995 May 11;332(19):1245-507708067
Cites: Stat Med. 2009 Jul 10;28(15):1982-9819452569
Cites: BMJ. 2011 Feb 22;342:d67121343207
Cites: Eur J Cardiovasc Prev Rehabil. 2004 Feb;11(1):48-5515167206
Cites: Am J Epidemiol. 1999 Mar 15;149(6):531-4010084242
Cites: Lancet Oncol. 2007 Apr;8(4):292-317431955
Cites: J Epidemiol Community Health. 2014 Jan;68(1):71-724166583
Cites: BMJ. 2015 Feb 10;350:h38425670624
Cites: Alcohol Clin Exp Res. 2008 Apr;32(4):645-5118241313
Cites: BMJ. 2014 Jul 10;349:g416425011450
Cites: Int J Cancer. 2012 Nov 1;131(9):2085-9322307919
Cites: J Stud Alcohol Drugs. 2016 Mar;77(2):185-9826997174
Cites: Acta Radiol Oncol. 1984;23(5):305-136095600
Cites: Int J Epidemiol. 2013 Dec;42(6):1772-9024415611
Cites: Stat Med. 2011 Feb 20;30(4):377-9921225900
Cites: Br J Cancer. 2004 Apr 5;90(7):1386-9115054460
Cites: Int J Epidemiol. 2015 Jun 10;:null26066328
Cites: BMJ. 2009 Jun 29;338:b239319564179
Cites: Eur J Epidemiol. 2011 Feb;26(2):81-9021267637
Cites: Addiction. 2006 Jul;101(7):959-7016771888
Cites: BMJ. 2015 Aug 18;351:h423826286216
Cites: J Stud Alcohol. 2003 Mar;64(2):278-8512713203
Cites: BMJ. 2011 Feb 22;342:d63621343206
Cites: BMJ. 1991 Sep 7;303(6802):553-61912885
Cites: J Epidemiol Community Health. 2006 Apr;60(4):345-5016537353
Cites: Arch Intern Med. 2006 Dec 11-25;166(22):2437-4517159008
PubMed ID
27807087 View in PubMed
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The association between alcohol consumption and mortality: the Swedish women's lifestyle and health study.

https://arctichealth.org/en/permalink/ahliterature137602
Source
Eur J Epidemiol. 2011 Feb;26(2):81-90
Publication Type
Article
Date
Feb-2011
Author
Gundula Behrens
Michael F Leitzmann
Sven Sandin
Marie Löf
Iris M Heid
Hans-Olov Adami
Elisabete Weiderpass
Author Affiliation
Department of Epidemiology and Preventive Medicine, Regensburg University Medical Center, Franz-Josef-Strauss-Allee 11, 93053 Regensburg, Germany. gundula.behrens@klinik.uni-regensburg.de
Source
Eur J Epidemiol. 2011 Feb;26(2):81-90
Date
Feb-2011
Language
English
Publication Type
Article
Keywords
Adult
Alcohol Drinking - mortality
Cardiovascular Diseases - mortality
Cohort Studies
Female
Follow-Up Studies
Humans
Middle Aged
Neoplasms - mortality
Proportional Hazards Models
Prospective Studies
Risk
Risk factors
Self Report
Sweden
Women's health
Abstract
Although light to moderate alcohol intake may reduce cardiovascular disease (CVD) mortality, the effect on total mortality requires further study, particularly among young and middle-aged women. We studied the association between alcohol consumption and mortality from all causes, from cancer, and from CVD in the Swedish Women's Lifestyle and Health Study, a cohort of 47,921 female residents of Sweden aged 30-49 years at baseline in 1991/1992 and followed up to 2006. We estimated the relative risk (RR) of mortality associated with alcohol intake using Cox regression adjusted for age, smoking, BMI, saturated fat intake, physical activity, and education. During 713,295 person-years of follow-up, 1,119 deaths occurred, including 158 deaths from CVD, 673 deaths from cancer, and 288 deaths from other causes. Compared with non-drinking, light to moderate drinking (0.1-19.9 g of alcohol per day) showed a statistically significant inverse association with total mortality (RR = 0.83, 95% CI = 0.71-0.98). Analyses of cause-specific mortality revealed an RR for CVD mortality of 0.69 (95% CI = 0.46-1.01) and an RR for cancer mortality of 0.92 (95% CI = 0.75-1.15). These results suggest that in younger women, a possibly beneficial effect of light to moderate drinking on future risk of mortality is limited to a prevention of CVD mortality but not cancer mortality.
PubMed ID
21267637 View in PubMed
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Changes in body mass index and waist circumference and concurrent mortality among Swedish women.

https://arctichealth.org/en/permalink/ahliterature284950
Source
Obesity (Silver Spring). 2017 Jan;25(1):215-222
Publication Type
Article
Date
Jan-2017
Author
Nina Roswall
Yingjun Li
Sven Sandin
Peter Ström
Hans-Olov Adami
Elisabete Weiderpass
Source
Obesity (Silver Spring). 2017 Jan;25(1):215-222
Date
Jan-2017
Language
English
Publication Type
Article
Keywords
Adult
Body mass index
Cardiovascular Diseases - mortality
Diet
European Continental Ancestry Group
Female
Follow-Up Studies
Humans
Life Style
Middle Aged
Neoplasms - mortality
Obesity - mortality
Proportional Hazards Models
Prospective Studies
Socioeconomic Factors
Surveys and Questionnaires
Sweden - epidemiology
Waist Circumference
Abstract
Most studies on obesity and mortality use a single anthropometric measure. Less is known about the effects of weight change on mortality. This study examined changes in body mass index (?BMI) and waist circumference (?WC) and subsequent all-cause and cause-specific mortality.
The study was conducted in the Women's Lifestyle and Health cohort, using self-reported anthropometric measures from 1991 to 1992 and 2003. Hazard ratios of mortality and 95% confidence intervals were calculated using Cox proportional hazards models. ?BMI and ?WC were examined in quartiles of absolute and relative change, with the second quartile (moderate gain) as the reference.
There was a higher risk of death in the first quartile of relative ?BMI: HR 1.28 (1.04-1.56). Absolute ?BMI suggested the same pattern, but the result was nonsignificant. ?WC was not associated with mortality. In cause-specific analyses, the association remained significant for cancer mortality only. In sensitivity analyses excluding the first 5 years of follow-up, the association was, however, attenuated.
This study found a higher risk of death among women in the first quartile of relative ?BMI compared with the second. It was driven by cancer mortality but may be ascribed to reverse causality. ?WC was not associated with mortality.
PubMed ID
27768253 View in PubMed
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Cohort Profile: The Swedish Women's Lifestyle and Health cohort.

https://arctichealth.org/en/permalink/ahliterature295341
Source
Int J Epidemiol. 2017 04 01; 46(2):e8
Publication Type
Journal Article
Research Support, Non-U.S. Gov't
Date
04-01-2017

Determinants of long-term weight change among middle-aged Swedish women.

https://arctichealth.org/en/permalink/ahliterature283956
Source
Obesity (Silver Spring). 2017 Feb;25(2):476-485
Publication Type
Article
Date
Feb-2017
Author
Darline El Reda
Peter Ström
Sven Sandin
Jin-Kyoung Oh
Hans-Olov Adami
Marie Löf
Elisabete Weiderpass
Source
Obesity (Silver Spring). 2017 Feb;25(2):476-485
Date
Feb-2017
Language
English
Publication Type
Article
Keywords
Adult
Alcohol Drinking
Exercise
Female
Humans
Life Style
Middle Aged
Obesity - prevention & control
Smoking
Smoking Cessation
Sweden
Weight Gain - physiology
Abstract
To describe the determinants of 12-year weight change among middle-aged women in Sweden.
In 1991/1992, 49,259 women across Sweden were recruited into a cohort. In 2003, 34,402 (73%) completed follow-up. Lifestyle and health characteristics including weight were collected, and 12-year weight change and substantial weight gain (=+5.0 kg) were calculated; association between baseline characteristics and odds ratios (OR) with 95% confidence intervals (CI) of substantial weight gain were estimated.
During the 12-year follow-up, 81% of women experienced weight gain. Being above average weight (64.5 kg) at baseline (OR =1.20, 95% CI: 1.14-1.26) and smoking 1 to 9 (OR?=?1.10, 95% CI: 1.01-1.20), 10 to 19 (OR?=?1.30, 95% CI: 1.21-1.39), or =20 cigarettes daily (OR?=?1.17, 95% CI: 1.04-1.32) increased a woman's odds of experiencing substantial weight gain (influenced by smoking cessation). In contrast, risk of substantial weight gain was reduced among women 45 to 50 years of age (OR?=?0.79, 95% CI: 0.73-0.85), women reporting high alcohol consumption (OR?=?0.90, 95% CI: 0.83-0.98), and those with medium (OR?=?0.93, 95% CI: 0.87-1.00) or high (OR 0.83, 95% CI: 0.77-0.90) physical activity levels.
The majority of women experienced weight gain during middle age. Population-specific determinants of weight gain should guide obesity prevention efforts.
PubMed ID
28063229 View in PubMed
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Endometrial cancer in relation to coffee, tea, and caffeine consumption: a prospective cohort study among middle-aged women in Sweden.

https://arctichealth.org/en/permalink/ahliterature264274
Source
Nutr Cancer. 2014;66(7):1132-43
Publication Type
Article
Date
2014
Author
Elisabete Weiderpass
Sven Sandin
Marie Lof
Jin-Kyoung Oh
Manami Inoue
Taichi Shimazu
Shoichiro Tsugane
Hans-Olov Adami
Source
Nutr Cancer. 2014;66(7):1132-43
Date
2014
Language
English
Publication Type
Article
Keywords
Adult
Body mass index
Caffeine - adverse effects
Coffee - adverse effects
Endometrial Neoplasms - epidemiology
Female
Follow-Up Studies
Humans
Middle Aged
Proportional Hazards Models
Prospective Studies
Risk factors
Sweden - epidemiology
Tea - adverse effects
Abstract
This study aimed to add to prospective data on the possible inverse association between coffee consumption and endometrial cancer risk, already supported by several case-control studies. Coffee and tea consumption and possible confounding factors were assessed among 42,270 women aged 30-49 years at enrollment in 1991-1992 in the Swedish Women's Lifestyle and Health cohort study, with complete follow-up through 2009. We calculated caffeine intake per day; Cox proportional hazard models were used to estimate multivariable relative risks (mRR) for endometrial cancer with 95% confidence intervals (CIs). One hundred forty-four endometrial cancers were diagnosed during follow-up. Women with and without endometrial cancer had a similar mean daily coffee consumption (549 vs. 547 g), tea consumption (104 vs. 115 g), and caffeine intake (405 vs. 406 mg). Compared to those consuming 3 cups had a mRR of 1.56 (95% CI: 0.94-2.59; P for trend = 0.17). Compared with the lowest tertile of caffeine intake, the highest tertile had a mRR of 1.32 (95% CI: 0.87-1.99; P for trend = 0.27). Our study provides no convincing evidence of an association between coffee consumption, tea consumption, or caffeine intake and endometrial cancer risk among middle-aged women.
PubMed ID
25181598 View in PubMed
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23 records – page 1 of 3.